37th Hemophilia Symposium Hamburg 2006: by I. Scharrer, Wolfgang Schramm

By I. Scharrer, Wolfgang Schramm

This booklet comprises the contribution to the thirty seventh Hemophilia Symposium, Hamburg 2006. the most issues are epidemiolgy, remedy of inhibitors in hemophiliacs, hemophilic arthropathy and synovitis, proper hemophilia remedy 2006, and pediatric hemostasiology. the quantity is rounded off via quite a few unfastened papers and posters on hemophilia, casuistics, and diagnostics.

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Additional info for 37th Hemophilia Symposium Hamburg 2006: Epidemiology;Treatment of Inhibitors in Hemophiliacs; Hemophilic Arthropathy and Synovitis; Relevant ... 2006; Pediatric Hemostasiology; Free Lectures

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Dr. , Dr. , Dr. , Frau Dr. , Dr. Ostschweizer Kinderspital, CH-St. Gallen Urbancik, W. Baxter, A-Wien Veldman, A. , Dipl. , Dr. , D-Markkleeberg Vogel, G. G. Universitätskliniken Mainz, D-Mainz von Auer, F. , Prof. , Dr. -W. , Dr. , Dr. St. , Dr. St. , Dr. Universitätskliniken, Institut für Exp. , Dr. H. Universitätskliniken, Kinder-Onkologie-, Hämatologie- und Immunologie, D-Düsseldorf Weiss, J. , Dr. SRH Fachkrankenhaus Neckargemünd, D-Neckargemünd Weller, D. , Dr. , Dr. Institut für Exp. , Frau Dr.

2006: 1. Type of factor substitute (Fig. 4) The differentiation shows a relevant shift in favor of the recombinant factor VIII products, 233 (71%) patients vs. plasma-derived factor VIII in 72 (22%) patients. Here the large group of patients without any selection is not equivalent to missing data but represents the fact that 189/516 patients (mostly mild disease) don’t need any treatment at all. 2. Annual amount of products The system allows to retrieve data for the amount of any product delivered to the patient for factor VIII or IX recombinant vs.

To address these questions, we developed a method to track FVIII-specific memory B cells in the blood of hemophilia A patients. This method is based on a technology that was recently described by Crotty et al. [7] for the detection of human memory B cells specific for viral antigens. To establish our method we used tetanus toxin as a control antigen and followed the kinetic of tetanus-specific memory B cells in the peripheral blood of healthy volunteers after vaccination. Fig. 1. Re-exposure of FVIII-specific memory B cells to FVIII and interaction with activated T cells at the same time cause re-stimulation and differentiation of FVIII-specific memory B cells into anti-FVIII antibody-producing plasma cells.

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